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Top 100 Observations Incompatible With The HIV-AIDS Construct
by John Kirkham
All of the observations below can be substantiated by
independent
research. How long can the HIV=AIDS=Death dogma be maintained
in the
face of so many scientific cracks?
1. AIDS occurs in the absense of HIV (65, 87), a new medical
definition (Idiopathic CD4+ T-cell lymphocytopenia) was
therefore
created.
2. HIV does not satisfy Koch's postulates, the criteria that
must be
met in order to prove that a microbe causes a disease (90)
3. Anti-HIV drugs, including protease inhibitors, destroy
T-cells (4-
10)
4. Septrin (also called Septra, Bactrim, Co-trimoxazole) and
anti-HIV
drugs destroy mitochondria (11,12)
5. The PCP (Pneumocystis Carinii pneumonia) fungus becomes
resistant
to Septrin (12)
6. Recreational drugs (heroin, poppers, crystal met, ecstasy,
cocaine) reduce CD4 cell numbers (13-18, 58, 66-68)
7. HIV positive patients recover after they stop taking drugs
(58)
8. Recreational drugs cause AIDS-defining diseases (see table
7 of 58)
9. Anti-HIV drugs cause AIDS-defining diseases (58)
10. Anti-HIV drugs inhibit human enzymes (11)
11. HIV positive Africans in dire poverty in Uganda and no
access to
anti-HIV drugs lived as long as HIV positives in the West who
took
anti-HIV drugs (33)
12. There are no comparative studies of survival in HIV
negatives and
combo-free HIV positive heterosexuals with no other risk
factors.
13. Only 38% of healthy long-term positives had ever used AZT
or
other nuleoside analogs compared with 94% of progressors (80)
14. Decreases in AIDS cases preceded the introduction of new
drug
treatments (Dec 1995) by three full years (see fig. 6 of 106)
15. Anti-HIV drugs have anti-microbial effects (49, 50, 10)
16. The introduction of AZT did not cause a decline in the
AIDS death
rate (105)
17. In the only long term trial of AZT (The Concorde study)
172
participants died, 169 while taking AZT, 3 while on placebo
(51)
18. Nucleoside analog drugs suppress/destroy the bone marrow
where
all immune system cells are born (26, 32, 111)
19. HIV+ children born to AZT treated mothers had a higher
probability of developing severe disease or severe
immunsuppression
(53)
20. "Drug holidays" recover immune responses
21. AZT caused the same transient increase in CD4 count in HIV
negatives as in HIV positives (55)
22. There are no controlled studies showing that AIDS occurs
in the
absense of all other possible non-HIV causal factors.
23. Long-living, healthy, drug-free HIV positives are mostly
ignored
by AIDS researchers
24. Apart from the early (fraudulent) AZT studies and the
Concorde
study no efficacy studies compare drugs with placebo
25. There are well documented, non-HIV causes for every AIDS
disease
26. The incidence of AIDS-defining diseases among Western
non-drug
users has not been shown to exceed national backgrounds (58)
27. Early AIDS coincided with the cumulative effects of
unprecedented, intense use of volatile nitrite (poppers) as a
aphrodisiac marketed almost exclusively to homosexuals (102)
28. AIDS can be treated effectively without anti-HIV drugs
(39-42,
112)
29. On average viral load overestimates infectious HIV by a
factor of
60,000 (21)
30. Even a PCR method that can detect 1 infected cell in
100000 found
very little HIV DNA in HIV positives (23)
31. HIV could not be cultured from people with a detectable
viral
load (19, 21)
32. HIV has never been properly isolated (20)
33. After many billions of dollars of research effort over 20
years,
HIV scientists still cannot explain how HIV causes AIDS.
34. After many billions of dollars of research effort over 20
years
there is no vaccine and no cure, there are only toxic drugs
35. There was no increase in HIV seroprevalence outside risk
groups
in the UK despite record STD rates and teenage pregnancy rates
(25)
36. HIV DNA was found to be constant from the time of
seroconversion
but CD4 count continually went down (29)
37. CD4 count goes down and viral load goes up while on the
anti-HIV
drugs.
38. AZT is hardly triphosphorylated by the body so it cannot
possibly
have an anti-HIV effect (30)
39. AZT has no effect on HIV DNA but makes viral load (HIV
RNA) go
down (31)
40. Research throughout the 1970s showed that retroviruses do
not
kill cells.
41. The probability of heterosexual transmission of HIV was
found to
be very low (1 in a 1000) (34)
42. HIV antibody tests can give repeated false positives and
seroreversions can occur (114-122)
43. HIV tests are sensitive to non-specific antibody binding
44. HIV tests involve an arbitrary dilution factor, everyone
tests
positive (because of non-specific antibody binding) if their
serum is
undiluted (104)
45. All the proteins used in the HIV test are associated with
retroviral genes that are found naturally (endogenous) in all
humans
(72)
46. Endogenous retroviruses can generate immune responses in
humans
(73, 74)
47. None of the HIV proteins tested for have been proven to
belong to
HIV (75)
48. There are over 60 different conditions, including
pregnancy, that
have been known to generate false positives on the HIV test
(91)
49. The Elisa, Western Blot and PCR tests for HIV all carry
disclaimers nullifying their detection of HIV
50. The criteria for HIV-positivity used in the antibody tests
varies
between countries and between organisations within a country
and can
produce indeterminate (neither positive or negative) results
(75,
109) The Western Blot HIV test, widely regarded as the most
accurate,
is not used in England and Wales because it is regarded as
inaccurate.
51. The viral load PCR primers were found to be non-specific
for "HIV" genetic sequences (35)
52. The viral load test gives false negatives (36)
53. The viral load test gives false positives (36, 113)
54. The viral load test has low reproducibility (36-38)
55. Direct measurements showed no correlation between viral
load and
CD4 count (43)
56. Many conditions cause reduced CD4 counts (86)
57. CD4 counts between 200 and 300 have been observed in
healthy HIV
negatives (87)
58. There are no studies comparing CD4 cell variations in
combo-free
HIV positives (with no risk factors) and HIV negatives.
59. According to the AIDS establishment, a heterosexual
AIDS "epidemic" of African origin started off in the
West as a
homosexual "epidemic"
60. In 1985 HIV incidence in Southern Africa was confined to
homosexuals who had been to the US and those who had had sex
with
them (88, 89).
61. The USA was found to be the world's most sexually
promiscuous
nation (27)
62. Condoms (made from polyisoprene) have holes in much larger
than
HIV (28, 110)
63. Reducing STD incidence in Africa did not reduce the rate
of HIV
seroconversion* (101)
64. Only a minute proportion of Africans have actually been
tested
for HIV, seroprevalence estimates are derived from
extrapolations
based on unrepresentative samples from maternity clinics.
65. In Africa a single positive ELISA test or even a single
"rapid"
(saliva/urine) test is considered proof of HIV infection,
"proof" in
the developed world requires a series of tests
66. HIV seroprevalence was found to be much lower in South
African
prisons than in the general population (1)
67. The vast majority of African "AIDS patients"
tested HIV negative
(44, 45)
68. In "AIDS ravaged" Zambia since 1980 the
population has increased
and even the rate of increase in population has increased!
(46)
69. In "AIDS ravaged" South Africa many coffin
makers are either
doing a slack trade or have gone out of business (47)
70. The total number of AIDS cases in Africa consists almost
entirely
of estimated cases rather than known, registered cases (54)
71. PCP is the typical AIDS defining disease in Western adults
but it
is almost entirely confined to young children in Africa (2,3)
72. There is no Western heterosexual AIDS epidemic
73. IVDUs who consistently used a clean needle exchange
program were
10.2 to 22.9 times MORE likely to test HIV positive than
non-users
(48)
74. Non-human primates "progress" to AIDS (SAIDS)
much quicker than
humans do (107)
75. SIV does not cause SAIDS in wild primate populations (108)
76. SIV seroprevalence is too low in wild primate populations
to
account for SIV resistance in these populations (22)
77. SIV seroprevalence in captive SIV naïve primate
populations was
found to be very low (22)
78. Until the early 1930s many thousands of European men
received
transplants from chimpanzees and did not get AIDS (62)
79. Uganda study showed HIV-positivity did not indicate a new
cause
of disease, only decreased mortality in HIV negatives (52)
80. One thousand medical staff a year accidentally contract
hepatitis
from needles yet by 1998 there were no documented cases of
surgeons
or emergency medical technicians/paramedics getting AIDS, or
even
HIV, from occupational exposure (58, Table 16 of 106)
81. All AIDS patients have lowered levels of glutathione, the
major
water soluble intracellular antioxidant (59, 60)
82. The antioxidant N-acetyl cysteine inhibits "HIV
replication" (61)
83. Reactive oxygen species are implicated in the induction of
HIV
expression and cell death (40)
84. Treatment with oxidising, mitogenic*** agents is necessary
for
HIV "isolation" from cell culture (56, 57)
85. Rectally deposited sperm can be immunosuppressive,
mitogenic,
oxidising and a stimulator of antigen production (94-100)
86. Low T-cell counts were shown to occur before HIV
seroconversion
and to predict seroconversion (92, 93)
87. HIV-like genetic sequences have been found in the HIV
negative
human genome (63)
88. Epitopes** of HIV regulatory proteins tat, rev and nef are
expressed in normal human tissue (71, 116)
89. Toxic intracellular stresses can create novel genetic
sequences
(64)
90. HIV showed over 40% variation in an essential gene
(protease)
sequence (103)
91. Foreign protein transfusions were found to be immune
suppressive
(79, 81, 84, 85)
92. Hemophiliacs can have hypergammaglobulinaemia which can
cause
false HIV positive test results (69)
93. Up to 99.9% of HIV genomes in plasma may be defective (70)
94. Mortality in hemophiliacs began to increase in exactly the
same
year they began taking AZT (81, 82)
95. The AIDS risk of hemophiliacs on AZT was 4.5 times higher,
and
mortality 2.4 times higher, than untreated controls (83)
96. Infectious HIV (a delicate virus) does not survive the
Factor
VIII preparation process (76-78)
97. HIV theorists have made incorrect predictions throughout
the HIV
era.
98. Corticosteroids and endogenous cortisol suppress cellular
immune
responses and cortisol destroys immature T-cells (24)
99. Effective cellular immunity relies upon nitric oxide gas
defence,
see for example Eur. J. Immunol. 2002, 32(5):1455-63
100. AIDS spreads non-exponentially, unlike infectious disease
(58)
References
(2) American J of
Respiratory and Critical Care Medicine 1994, 149(6):1591-1596
(3) Central African J of Medicine 1999, 45:127-8 (4) J Virol
2002, 76
(12):5966-73 (5) J Biol Chem 1989, 264:6127-33 (6)
Antimicrobial
Agents and Chemotherapy 1990, 34:637-641 (7) Antiviral
Chemistry and
Chemotherapy 1991, 2:125-132 (8) AIDS 1989, 3:417-422
(9) NEJM 1987, 317:192-197 (10) Physicians Desk Reference 1999
(11)
Nature Medicine 1995, 1(5):417-422
(12)
(13)Pharmacotherapy 1984, 4:284-291
(14) Cancer Research 1983,
43:1365-
1371
(15) Lancet 1982, Feb 20, 412-416 (16) AIDS 1991, 5:35-41 (17)
Annals
NY Acad. Sci. 1987, 496:711-21 (18) Life Sciences 2001,
69:2931-2941
(19) NEJM 1995, 332:201-208 (20) Virol. 1997, 230:125-133 (21)
Science 1993, 259:1749-1754
(22) http://forums.about.com/innocuous/messages?msg=47.1
(23) J Virol
1990, 64: 864-872 (24) Medical Hypothesis 1996, 46:551-555
(25) The
Times (UK) June 2nd 2001 (26) Adverse Drug Reaction Bulletin
1996,
178:675-8.
(27) Durex Global Sex Survey 2001,
(28) Rubber
Chemistry and Technology, 1989, 62(4):683-697 (see page 692)
(29) J.
AIDS 1994, 7:381-388 (30) Current Medical Research and Opinion
1999,
Vol. 15, supplement 1 (31) J. AIDS 1991, 4:766-9
(33) http://healtoronto.com/richards.html
(34) American J.
Epidemiology 1997, 146(4):350-357 (35) AIDS 1998, 12:2076-2077
(36)
Annals of Internal Medicine 1996, 124:803-815 (37) J. of AIDS
and
Human Retrovirology 1997, 15(2):174-5 (38) J. AIDS 1992,
5(9):872-877
(39) Proc. Nat. Acad. Sci. USA 1997, 94:1967-1972 (40) Medical
Hypothesis 1993, 40(2):85-92 (41) Trans. Assoc. Am. Phys.
1984, 97:70-
79 (42) Proc. Soc. Exp. Biol. Med. 1997, 216:201-210 (43)
Nature
Medicine 1999, 5(1):83-89 (see fig. 4b) (44) J. AIDS 1994,
7(8):876-
877
(45) Lancet 1992, 340:971-972 (46)
http://esa.un.org/
(47)
(48) American J.
Epidemiology 1997, 146(12):994-1002 (see table 5) (49) J. of
Infectious Diseases 2000, 181:1629-1634 (50) J. of Infectious
Diseases 1999, 180:448-453 (51) Lancet 1994, 343:871-881 (52)
Lancet
1994, 343:1021-1023 (53) AIDS 1999, 13(8):927-33 (54) See
tabulated
data in the annex to the WHO Global Report 1998 (55) AIDS
1996, 10
(12):1444-5 (56) Science 1986, 231:850-853 (57) Nature 1986,
319:10-11
(58) Genetica 1998, 104:85-132 (59) FASEB J. 1997,
11:1077-1089 (60)
Proc. Natl. Acad. Sci. USA 1997, 94:1967-1972 (61) Proc. Natl.
Acad.
Sci. USA 1991, 88:986-990 (62) Hamilton D. The Monkey Gland
Affair,
Chatto and Windus Ltd., London 1986 (63) J. Virol. 1992,
66:2170-2179
(64) Clin. Diagn. Lab. Immunol. 1992, 6(3):330-335 (65)
Biotechnology
1993, 11:955-956 (66) AIDS 1987, 1:105-111
(67) American J. Epidemiology 1993, 137(9):989-1000 (68) Clin.
Immunol. Immunopathol. 1994, 70:245-250 (69) Isr. J. Med. Sci.
1991,
27:557-561 (70) Nature 1993, 364:291 (71) Am. J. Pathol. 1992,
141:1209-1216 (72) JAMA 1988, 260(5):674-679 (73)
Immunological
Reviews 1996, 152:193-236 (74) Proc. Natl. Acad. Sci. USA
1996,
93:5177-5184 (75) Biotechnology 1993, 11:696-707
(76) CDC Fact sheet on HIV transmission January, 1994 (77)
JAMA 1989,
261:1275 (78) J. AIDS 1992, 5:822-828 (79) Ann. Int. Med.
1985,
103:723-726 (80) AIDS 1994, 8:1123 (81) Genetica 1995,
95:51-70 (82)
Lancet 1995, 346:1371-1372 (83) Lancet 1994, 344:791-792, see
table
on page 791 (84) NEJM 1984, 322:941-949 (85) Am. J. Hematol.
1985,
20:1-6 (86)
(87) J.
Antimicrobial Therapy 1996, 37(Suppl. B):171-83 (88) S. Afr.
Med. J.
1985, 68(8):617-8
(89) NEJM 1985 312(19):1257-8 (90)
(91) http://www.virusmyth.net/aids/data/cjtestfp.htm
(92) J. AIDS
1993, 6:820-822 (93) Epidemiology 1990, 1:453-459 (94) JAMA
1984,
251:237 (95) Br. Med. J. 1983, 286:1651 (96) NEJM 1983,
308:1181 (97)
Fed. Proc. 1983, 42:1334 (98) J. Exp. Med. 1982, 155:1719
(99) Immunol. Today 1984, 5:357 (100) Theor. Biol. 1982,
96:741 (101)
Lancet 1999, 353:525-535 (102) Kitzerow M. The AIDS
Indictment, MRKCO
Publishing, Chicago, 2000. See http://www.aidsindictment.com
(103)
Nature Medicine 1996, 4(7):753-759
(104)
(105) http://www.cdc.gov/hiv/stats/hasrsupp81/fig9.htm
(106) CDC
HIV/AIDS Surveillance Report, Year-end 1997 (Vol. 9 No. 2)
(107)
Science 1990, 248:1109-1112 (108) J. Virol. 2001, 75:2262-2275
(109)
The Perth Group, Mother to Child Transmission of HIV and its
Prevention with AZT and Nevirapine, ISBN 1876763728, page 5.
(110)
(111)
http://www.virusmyth.net/aids/data/dchaart.htm#adverse_nukes
(112)
(114) Annals of
Internal Medicine 1988, 108:785-90 (115) Annals of Internal
Medicine
1985, 103:545-7 (116) AIDS 1992, 6:1547-48 (117) JAMA 1992,
268
(8):1015-1017 (118) Lancet 1992; 339:1548
(119) Lancet 1993, 342:1458-1459 (120) Absts Ist National
Conf. Human
Retroviruses. Abst. 1993, #86, p. 71.
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