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“The only thing necessary for these diseases to the triumph is for good people and governments to do nothing.”

       
     

Top 100 Observations Incompatible With The HIV-AIDS Construct

by John Kirkham

All of the observations below can be substantiated by independent
research. How long can the HIV=AIDS=Death dogma be maintained in the
face of so many scientific cracks?

1. AIDS occurs in the absense of HIV (65, 87), a new medical
definition (Idiopathic CD4+ T-cell lymphocytopenia) was therefore
created.
2. HIV does not satisfy Koch's postulates, the criteria that must be
met in order to prove that a microbe causes a disease (90)
3. Anti-HIV drugs, including protease inhibitors, destroy T-cells (4-
10)
4. Septrin (also called Septra, Bactrim, Co-trimoxazole) and anti-HIV
drugs destroy mitochondria (11,12)
5. The PCP (Pneumocystis Carinii pneumonia) fungus becomes resistant
to Septrin (12)
6. Recreational drugs (heroin, poppers, crystal met, ecstasy,
cocaine) reduce CD4 cell numbers (13-18, 58, 66-68)
7. HIV positive patients recover after they stop taking drugs (58)
8. Recreational drugs cause AIDS-defining diseases (see table 7 of 58)
9. Anti-HIV drugs cause AIDS-defining diseases (58)
10. Anti-HIV drugs inhibit human enzymes (11)
11. HIV positive Africans in dire poverty in Uganda and no access to
anti-HIV drugs lived as long as HIV positives in the West who took
anti-HIV drugs (33)
12. There are no comparative studies of survival in HIV negatives and
combo-free HIV positive heterosexuals with no other risk factors.
13. Only 38% of healthy long-term positives had ever used AZT or
other nuleoside analogs compared with 94% of progressors (80)
14. Decreases in AIDS cases preceded the introduction of new drug
treatments (Dec 1995) by three full years (see fig. 6 of 106)
15. Anti-HIV drugs have anti-microbial effects (49, 50, 10)
16. The introduction of AZT did not cause a decline in the AIDS death
rate (105)
17. In the only long term trial of AZT (The Concorde study) 172
participants died, 169 while taking AZT, 3 while on placebo (51)
18. Nucleoside analog drugs suppress/destroy the bone marrow where
all immune system cells are born (26, 32, 111)
19. HIV+ children born to AZT treated mothers had a higher
probability of developing severe disease or severe immunsuppression
(53)
20. "Drug holidays" recover immune responses
21. AZT caused the same transient increase in CD4 count in HIV
negatives as in HIV positives (55)
22. There are no controlled studies showing that AIDS occurs in the
absense of all other possible non-HIV causal factors.
23. Long-living, healthy, drug-free HIV positives are mostly ignored
by AIDS researchers
24. Apart from the early (fraudulent) AZT studies and the Concorde
study no efficacy studies compare drugs with placebo
25. There are well documented, non-HIV causes for every AIDS disease
26. The incidence of AIDS-defining diseases among Western non-drug
users has not been shown to exceed national backgrounds (58)
27. Early AIDS coincided with the cumulative effects of
unprecedented, intense use of volatile nitrite (poppers) as a
aphrodisiac marketed almost exclusively to homosexuals (102)
28. AIDS can be treated effectively without anti-HIV drugs (39-42,
112)
29. On average viral load overestimates infectious HIV by a factor of
60,000 (21)
30. Even a PCR method that can detect 1 infected cell in 100000 found
very little HIV DNA in HIV positives (23)
31. HIV could not be cultured from people with a detectable viral
load (19, 21)
32. HIV has never been properly isolated (20)
33. After many billions of dollars of research effort over 20 years,
HIV scientists still cannot explain how HIV causes AIDS.
34. After many billions of dollars of research effort over 20 years
there is no vaccine and no cure, there are only toxic drugs
35. There was no increase in HIV seroprevalence outside risk groups
in the UK despite record STD rates and teenage pregnancy rates (25)
36. HIV DNA was found to be constant from the time of seroconversion
but CD4 count continually went down (29)
 

    

37. CD4 count goes down and viral load goes up while on the anti-HIV
drugs.
38. AZT is hardly triphosphorylated by the body so it cannot possibly
have an anti-HIV effect (30)
39. AZT has no effect on HIV DNA but makes viral load (HIV RNA) go
down (31)
40. Research throughout the 1970s showed that retroviruses do not
kill cells.
41. The probability of heterosexual transmission of HIV was found to
be very low (1 in a 1000) (34)
42. HIV antibody tests can give repeated false positives and
seroreversions can occur (114-122)
43. HIV tests are sensitive to non-specific antibody binding
44. HIV tests involve an arbitrary dilution factor, everyone tests
positive (because of non-specific antibody binding) if their serum is
undiluted (104)
45. All the proteins used in the HIV test are associated with
retroviral genes that are found naturally (endogenous) in all humans
(72)
46. Endogenous retroviruses can generate immune responses in humans
(73, 74)
47. None of the HIV proteins tested for have been proven to belong to
HIV (75)
48. There are over 60 different conditions, including pregnancy, that
have been known to generate false positives on the HIV test (91)
49. The Elisa, Western Blot and PCR tests for HIV all carry
disclaimers nullifying their detection of HIV
50. The criteria for HIV-positivity used in the antibody tests varies
between countries and between organisations within a country and can
produce indeterminate (neither positive or negative) results (75,
109) The Western Blot HIV test, widely regarded as the most accurate,
is not used in England and Wales because it is regarded as inaccurate.
51. The viral load PCR primers were found to be non-specific
for "HIV" genetic sequences (35)
52. The viral load test gives false negatives (36)
53. The viral load test gives false positives (36, 113)
54. The viral load test has low reproducibility (36-38)
55. Direct measurements showed no correlation between viral load and
CD4 count (43)
56. Many conditions cause reduced CD4 counts (86)
57. CD4 counts between 200 and 300 have been observed in healthy HIV
negatives (87)
58. There are no studies comparing CD4 cell variations in combo-free
HIV positives (with no risk factors) and HIV negatives.
59. According to the AIDS establishment, a heterosexual
AIDS "epidemic" of African origin started off in the West as a
homosexual "epidemic"
60. In 1985 HIV incidence in Southern Africa was confined to
homosexuals who had been to the US and those who had had sex with
them (88, 89).
61. The USA was found to be the world's most sexually promiscuous
nation (27)
62. Condoms (made from polyisoprene) have holes in much larger than
HIV (28, 110)
63. Reducing STD incidence in Africa did not reduce the rate of HIV
seroconversion* (101)
64. Only a minute proportion of Africans have actually been tested
for HIV, seroprevalence estimates are derived from extrapolations
based on unrepresentative samples from maternity clinics.
65. In Africa a single positive ELISA test or even a single "rapid"
(saliva/urine) test is considered proof of HIV infection, "proof" in
the developed world requires a series of tests
66. HIV seroprevalence was found to be much lower in South African
prisons than in the general population (1)
67. The vast majority of African "AIDS patients" tested HIV negative
(44, 45)
68. In "AIDS ravaged" Zambia since 1980 the population has increased
and even the rate of increase in population has increased! (46)
69. In "AIDS ravaged" South Africa many coffin makers are either
doing a slack trade or have gone out of business (47)
70. The total number of AIDS cases in Africa consists almost entirely
of estimated cases rather than known, registered cases (54)
71. PCP is the typical AIDS defining disease in Western adults but it
is almost entirely confined to young children in Africa (2,3)
72. There is no Western heterosexual AIDS epidemic
73. IVDUs who consistently used a clean needle exchange program were
10.2 to 22.9 times MORE likely to test HIV positive than non-users
(48)
74. Non-human primates "progress" to AIDS (SAIDS) much quicker than
humans do (107)
75. SIV does not cause SAIDS in wild primate populations (108)
76. SIV seroprevalence is too low in wild primate populations to
account for SIV resistance in these populations (22)
77. SIV seroprevalence in captive SIV naïve primate populations was
found to be very low (22)
78. Until the early 1930s many thousands of European men received
transplants from chimpanzees and did not get AIDS (62)
79. Uganda study showed HIV-positivity did not indicate a new cause
of disease, only decreased mortality in HIV negatives (52)
80. One thousand medical staff a year accidentally contract hepatitis
from needles yet by 1998 there were no documented cases of surgeons
or emergency medical technicians/paramedics getting AIDS, or even
HIV, from occupational exposure (58, Table 16 of 106)
81. All AIDS patients have lowered levels of glutathione, the major
water soluble intracellular antioxidant (59, 60)
82. The antioxidant N-acetyl cysteine inhibits "HIV replication" (61)
83. Reactive oxygen species are implicated in the induction of HIV
expression and cell death (40)
84. Treatment with oxidising, mitogenic*** agents is necessary for
HIV "isolation" from cell culture (56, 57)
85. Rectally deposited sperm can be immunosuppressive, mitogenic,
oxidising and a stimulator of antigen production (94-100)
86. Low T-cell counts were shown to occur before HIV seroconversion
and to predict seroconversion (92, 93)
87. HIV-like genetic sequences have been found in the HIV negative
human genome (63)
88. Epitopes** of HIV regulatory proteins tat, rev and nef are
expressed in normal human tissue (71, 116)
89. Toxic intracellular stresses can create novel genetic sequences
(64)
90. HIV showed over 40% variation in an essential gene (protease)
sequence (103)
91. Foreign protein transfusions were found to be immune suppressive
(79, 81, 84, 85)
92. Hemophiliacs can have hypergammaglobulinaemia which can cause
false HIV positive test results (69)
93. Up to 99.9% of HIV genomes in plasma may be defective (70)
94. Mortality in hemophiliacs began to increase in exactly the same
year they began taking AZT (81, 82)
95. The AIDS risk of hemophiliacs on AZT was 4.5 times higher, and
mortality 2.4 times higher, than untreated controls (83)
96. Infectious HIV (a delicate virus) does not survive the Factor
VIII preparation process (76-78)
 

    

97. HIV theorists have made incorrect predictions throughout the HIV
era.
98. Corticosteroids and endogenous cortisol suppress cellular immune
responses and cortisol destroys immature T-cells (24)
99. Effective cellular immunity relies upon nitric oxide gas defence,
see for example Eur. J. Immunol. 2002, 32(5):1455-63
100. AIDS spreads non-exponentially, unlike infectious disease (58)

References

 (2) American J of
Respiratory and Critical Care Medicine 1994, 149(6):1591-1596
(3) Central African J of Medicine 1999, 45:127-8 (4) J Virol 2002, 76
(12):5966-73 (5) J Biol Chem 1989, 264:6127-33 (6) Antimicrobial
Agents and Chemotherapy 1990, 34:637-641 (7) Antiviral Chemistry and
Chemotherapy 1991, 2:125-132 (8) AIDS 1989, 3:417-422
(9) NEJM 1987, 317:192-197 (10) Physicians Desk Reference 1999 (11)
Nature Medicine 1995, 1(5):417-422
(12)

 (13)Pharmacotherapy 1984, 4:284-291

(14) Cancer Research 1983, 43:1365-
1371
(15) Lancet 1982, Feb 20, 412-416 (16) AIDS 1991, 5:35-41 (17) Annals
NY Acad. Sci. 1987, 496:711-21 (18) Life Sciences 2001, 69:2931-2941
(19) NEJM 1995, 332:201-208 (20) Virol. 1997, 230:125-133 (21)
Science 1993, 259:1749-1754
(22) http://forums.about.com/innocuous/messages?msg=47.1 (23) J Virol
1990, 64: 864-872 (24) Medical Hypothesis 1996, 46:551-555 (25) The
Times (UK) June 2nd 2001 (26) Adverse Drug Reaction Bulletin 1996,
178:675-8.
(27) Durex Global Sex Survey 2001,
(28) Rubber
Chemistry and Technology, 1989, 62(4):683-697 (see page 692) (29) J.
AIDS 1994, 7:381-388 (30) Current Medical Research and Opinion 1999,
Vol. 15, supplement 1 (31) J. AIDS 1991, 4:766-9
(33) http://healtoronto.com/richards.html (34) American J.
Epidemiology 1997, 146(4):350-357 (35) AIDS 1998, 12:2076-2077 (36)
Annals of Internal Medicine 1996, 124:803-815 (37) J. of AIDS and
Human Retrovirology 1997, 15(2):174-5 (38) J. AIDS 1992, 5(9):872-877
(39) Proc. Nat. Acad. Sci. USA 1997, 94:1967-1972 (40) Medical
Hypothesis 1993, 40(2):85-92 (41) Trans. Assoc. Am. Phys. 1984, 97:70-
79 (42) Proc. Soc. Exp. Biol. Med. 1997, 216:201-210 (43) Nature
Medicine 1999, 5(1):83-89 (see fig. 4b) (44) J. AIDS 1994, 7(8):876-
877
(45) Lancet 1992, 340:971-972 (46) http://esa.un.org/ (47)
 (48) American J.
Epidemiology 1997, 146(12):994-1002 (see table 5) (49) J. of
Infectious Diseases 2000, 181:1629-1634 (50) J. of Infectious
Diseases 1999, 180:448-453 (51) Lancet 1994, 343:871-881 (52) Lancet
1994, 343:1021-1023 (53) AIDS 1999, 13(8):927-33 (54) See tabulated
data in the annex to the WHO Global Report 1998 (55) AIDS 1996, 10
(12):1444-5 (56) Science 1986, 231:850-853 (57) Nature 1986, 319:10-11
(58) Genetica 1998, 104:85-132 (59) FASEB J. 1997, 11:1077-1089 (60)
Proc. Natl. Acad. Sci. USA 1997, 94:1967-1972 (61) Proc. Natl. Acad.
Sci. USA 1991, 88:986-990 (62) Hamilton D. The Monkey Gland Affair,
Chatto and Windus Ltd., London 1986 (63) J. Virol. 1992, 66:2170-2179
(64) Clin. Diagn. Lab. Immunol. 1992, 6(3):330-335 (65) Biotechnology
1993, 11:955-956 (66) AIDS 1987, 1:105-111
(67) American J. Epidemiology 1993, 137(9):989-1000 (68) Clin.
Immunol. Immunopathol. 1994, 70:245-250 (69) Isr. J. Med. Sci. 1991,
27:557-561 (70) Nature 1993, 364:291 (71) Am. J. Pathol. 1992,
141:1209-1216 (72) JAMA 1988, 260(5):674-679 (73) Immunological
Reviews 1996, 152:193-236 (74) Proc. Natl. Acad. Sci. USA 1996,
93:5177-5184 (75) Biotechnology 1993, 11:696-707
(76) CDC Fact sheet on HIV transmission January, 1994 (77) JAMA 1989,
261:1275 (78) J. AIDS 1992, 5:822-828 (79) Ann. Int. Med. 1985,
103:723-726 (80) AIDS 1994, 8:1123 (81) Genetica 1995, 95:51-70 (82)
Lancet 1995, 346:1371-1372 (83) Lancet 1994, 344:791-792, see table
on page 791 (84) NEJM 1984, 322:941-949 (85) Am. J. Hematol. 1985,
20:1-6 (86)  (87) J.
Antimicrobial Therapy 1996, 37(Suppl. B):171-83 (88) S. Afr. Med. J.
1985, 68(8):617-8
(89) NEJM 1985 312(19):1257-8 (90)
(91) http://www.virusmyth.net/aids/data/cjtestfp.htm (92) J. AIDS
1993, 6:820-822 (93) Epidemiology 1990, 1:453-459 (94) JAMA 1984,
251:237 (95) Br. Med. J. 1983, 286:1651 (96) NEJM 1983, 308:1181 (97)
Fed. Proc. 1983, 42:1334 (98) J. Exp. Med. 1982, 155:1719
(99) Immunol. Today 1984, 5:357 (100) Theor. Biol. 1982, 96:741 (101)
Lancet 1999, 353:525-535 (102) Kitzerow M. The AIDS Indictment, MRKCO
Publishing, Chicago, 2000. See http://www.aidsindictment.com (103)
Nature Medicine 1996, 4(7):753-759
(104)
(105) http://www.cdc.gov/hiv/stats/hasrsupp81/fig9.htm (106) CDC
HIV/AIDS Surveillance Report, Year-end 1997 (Vol. 9 No. 2) (107)
Science 1990, 248:1109-1112 (108) J. Virol. 2001, 75:2262-2275 (109)
The Perth Group, Mother to Child Transmission of HIV and its
Prevention with AZT and Nevirapine, ISBN 1876763728, page 5.
(110)
  (111)
http://www.virusmyth.net/aids/data/dchaart.htm#adverse_nukes
(112)
 (114) Annals of
Internal Medicine 1988, 108:785-90 (115) Annals of Internal Medicine
1985, 103:545-7 (116) AIDS 1992, 6:1547-48 (117) JAMA 1992, 268
(8):1015-1017 (118) Lancet 1992; 339:1548
(119) Lancet 1993, 342:1458-1459 (120) Absts Ist National Conf. Human
Retroviruses. Abst. 1993, #86, p. 71.



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